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Furthermore, hyperketonemia is known to trigger ketoacidosis, but BHB or KD are used to improve metabolism or epilepsy, how to select the appropriate concentration of BHB without inducing ketoacidosis is still unknown
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Thus, DNA methylation may act as a critical mediator, integrating environmental signals such as inflammation and oxidative stress, and ultimately influencing the gene expression pathways involved in AMD pathogenesis (Hunter et al., 2012)
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